Traffic

杂志介绍

Traffic杂志介绍

《Traffic》是一本以English为主的未开放获取国际优秀期刊，中文名称交通，本刊主要出版、报道生物学-CELL BIOLOGY领域的研究动态以及在该领域取得的各方面的经验和科研成果，介绍该领域有关本专业的最新进展，探讨行业发展的思路和方法，以促进学术信息交流，提高行业发展。该刊已被国际权威数据库SCIE收录，为该领域相关学科的发展起到了良好的推动作用，也得到了本专业人员的广泛认可。该刊最新影响因子为3.6，最新CiteScore 指数为8.1。

本刊近期中国学者发表的论文主要有：

• Pseudoginsenoside-F11 alleviates oligomeric β-amyloid-induced endosome-lysosome defects in microglia.

  Author: Yao XC¹, Xue X², Zhang HT¹, Zhu MM¹, Yang XW¹, Wu CF¹, Yang JY¹.

• Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)/Cas9-mediated kif15 mutations accelerate axonal outgrowth during neuronal development and regeneration in zebrafish.

  Author: Dong Z¹, Wu S¹, Zhu C¹, Wang X², Li Y¹, Chen X¹, Liu D¹, Qiang L³, Baas PW³, Liu M¹.

• Mechanisms of the anterograde trafficking of GPCRs: Regulation of AT1R transport by interacting proteins and motifs.

  Author: Zhang M^1,2, Wu G².

• Protein kinase A-mediated phosphorylation of naked cuticle homolog 2 stimulates cell-surface delivery of transforming growth factor-α for epidermal growth factor receptor transactivation.

  Author: Cao Z^1,2, Singh B^1,2,3, Li C^4,5, Markham NO^1,2, Carrington LJ¹, Franklin JL^1,2,3,6, Graves-Deal R^1,2, Kennedy EJ⁷, Goldenring JR^2,3,6,8, Coffey RJ^1,2,3,6.

英文介绍

Traffic杂志英文介绍

Traffic encourages and facilitates the publication of papers in any field relating to intracellular transport in health and disease. Traffic papers span disciplines such as developmental biology, neuroscience, innate and adaptive immunity, epithelial cell biology, intracellular pathogens and host-pathogen interactions, among others using any eukaryotic model system. Areas of particular interest include protein, nucleic acid and lipid traffic, molecular motors, intracellular pathogens, intracellular proteolysis, nuclear import and export, cytokinesis and the cell cycle, the interface between signaling and trafficking or localization, protein translocation, the cell biology of adaptive an innate immunity, organelle biogenesis, metabolism, cell polarity and organization, and organelle movement.

All aspects of the structural, molecular biology, biochemistry, genetics, morphology, intracellular signaling and relationship to hereditary or infectious diseases will be covered. Manuscripts must provide a clear conceptual or mechanistic advance. The editors will reject papers that require major changes, including addition of significant experimental data or other significant revision.

Traffic will consider manuscripts of any length, but encourages authors to limit their papers to 16 typeset pages or less.